Giuseppe Michieli<p>Modeling suggests <a href="https://mstdn.science/tags/SARS" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>SARS</span></a>-CoV-2 <a href="https://mstdn.science/tags/rebound" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>rebound</span></a> after <a href="https://mstdn.science/tags/nirmatrelvir" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>nirmatrelvir</span></a>-ritonavir <a href="https://mstdn.science/tags/treatment" class="mention hashtag" rel="nofollow noopener noreferrer" target="_blank">#<span>treatment</span></a> is driven by target cell preservation coupled with incomplete viral clearance, <a href="https://etidiohnew.blogspot.com/2025/02/modeling-suggests-sars-cov-2-rebound.html" rel="nofollow noopener noreferrer" translate="no" target="_blank"><span class="invisible">https://</span><span class="ellipsis">etidiohnew.blogspot.com/2025/0</span><span class="invisible">2/modeling-suggests-sars-cov-2-rebound.html</span></a></p>